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This likely reflects the ability of sulphonylureas to promote membrane depolarization and calcium entry, which in turn facilitates activation of Ca 2 dependent metabolic enzymes such as mitochondrial dehydrogenases and glycogen phosphorylase 48, 49 tamoxifen endometrial Since peripheral infiltration of inflammatory components into the brain is thought to contribute to the late stage sequelae in AD and other tauopathies 82 85, our findings suggest that even late stage interventions targeting tau could help maintain BBB integrity and reduce the vascular contributions to cognitive impairment and dementia that occur in these diseases